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Moreover, a component of endothelial dysfunction has been proposed, which can contribute to abnormal vessel reactivity. However, testosterone in physiologic doses may even be helpful in sufferers with angina , have larger ischemic thresholds and improved high quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and increases coronary blood move in men with established coronary artery illness . Therefore, an interplay of endogenous components of the patient’s danger profile in addition to the testosterone dosing could additionally be problems with whether antagonistic occasions occur.

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We report a case of an acute ST-segment elevation myocardial infarction in a affected person with traditional cardiac danger components using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this patient also had polycythemia, probably secondary to high-dose testosterone. The affected person underwent profitable percutaneous intervention of the best coronary artery. Physicians and sufferers ought to pay consideration to the scientific consequences of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).

Anabolic Steroids, Acute Myocardial Infarction And Polycythemia: A Case Report And Evaluate Of The Literature

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